Head Injuries
- can involve skull fractures, hemorrhage and edema, or direct injury to brain
tissue
- full range of degree of damage
- bone fragments from skull can cause severe damage
- inability of skull to expand increasing intracranial pressure (pressure
within the skull)
- may be closed or open injuries
- fractures may be linear, comminuted, compound, depressed, basilar (at base
of skull)
- contrecoup injury, usually causing minor damage, can occur contralateral
to site of injury (opposite side of the skull)
concussions
- reversible dysfunction from mild blow
- usually full recovery in 24 hrs
contusion
- bruising of brain tissue
- possibility of residual damage
Hematoma (internal bleeding) can occur as
a result of head trauma:
epidural hematoma
- bleeding between dura and skull
subdural hematoma
- bleeding between dura and arachnoid
subarachnoid hemorrhage
- space between arachnoid and pia
intracerebral hematoma
- may develop days after injury
Signs and symptoms of head trauma:
- seizures (focal and general)
- cranial nerve impairment
- particularly with basilar fractures
- otorrhea, rhinorrhea (leaking CSF from ears, nose)
- fever (hypothalamic/cranial)
Herniation syndromes
- Cingulate (Subfalcine) Herniation
- The distal anterior cerebral artery (supplies motor cortex of leg) can be compressed causing contralateral leg weakness
- Uncal (Unilateral Transtentorial) Herniation
- Unilateral mass forces the ipsilateral (same side) temporal lobe through the tentorium cerebelli
- Contralateral upper motor neuron signs
- Ipsilateral third nerve compression
- Contralateral Upper Brainstem compressed
- Cerebrospinal Fluid (CSF) flow interrupted
- Abnormal respirations
- Central Herniation
- Supratentorial lesion forces the diencephalon centrally through the tentorium cerebelli
- Compresses the upper midbrain, lateral pons and medulla
- Early:
- erratic respirations
- small reactive pupils
- increased limb tone
- Babinski sign
- Decorticate rigidity (loss of higher motor control; flexion due to rubrospinal system intact)
- Decerebrate posturing (later) - dysfunction at red nucleus, extension due to reticulo- & vestibulospinal tracts intact
- Fixed and dilated pupils
- Tonsillar Herniation
- If posterior fossa pressure is high, the cerebellar tonsils can herniate through the foramen magnum of the skull.
- Loss of consciousness, as well as compromised cardiovascular and respiratory centres leading to labile BP and ireegular breathing.
Cushing Reflex
- The classical Cushing's reflex is a late event and often occurs pre-mortem
- Relative brain ischemia, causing systemic hypertension due to sympathetic outflow activation, and thereby producing bradycardia by feedback from the carotid baroreceptors